INTERNATIONAL INSTITUTE OF BIOPHYSICS
Conference on Biophotons 1999
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CARCINOGENESIS IN TERMS OF BIOPHOTONS

Fritz-Albert Popp

International Institute of Biophysics, Ehemalige Raketenstation, Kapellener Straße D-41472 Neuss

Fax: 02182 825132, E-mail: iib@lifescientists.de

About 25 years ago the author found significant correlations between transition momenta and energy levels of the lowest singlet - and triplet states of 37 polycyclic hydrocarbons (like Benzo(a)pyren and Benzo(e)pyren) and their carcinogenic activity [1,2]. Based on this connection he proposed that carcinogenic substances absorb light within the tissues which usually works for cell growth regulation and photorepair [1,2,3], disturbing thus the growth rate control. Up to now he maintains this proposal, putting into question the hypothesis that metabolism of the compounds is a necessary step of their activation. Just opposite, his statement of metabolic activity for inactivating carcinogenic risk has never been disproved.

With increasing insight into the correlations between malignant cell growth and biophoton emission and delayed luminescence [4-6], a theory could be developed where the basic regulatory function of cell growth and differentiation is the degree of coherence of the biophoton field within the organism under investigation [7.8]. Actually, it turns out that a nonlinear differential equation that describes quantitatively the growth rate in normal tissues explains tumor growth as well, as soon as the term describing the mean value of the coherence time within the tissue drops down to a definite threshold. This relation can become subject of examination by means of biophoton measurements.

The basic mechanism behind this dependence may be traced back to a negative feed back loop between the occupation probability of excited states of DNA and the coherence of the biophoton field.

References:

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